🗊Презентация Tissue repair. Regeneration and Reparation

Категория: Здоровье и Медицина
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Tissue repair. Regeneration and Reparation, слайд №1Tissue repair. Regeneration and Reparation, слайд №2Tissue repair. Regeneration and Reparation, слайд №3Tissue repair. Regeneration and Reparation, слайд №4Tissue repair. Regeneration and Reparation, слайд №5Tissue repair. Regeneration and Reparation, слайд №6Tissue repair. Regeneration and Reparation, слайд №7Tissue repair. Regeneration and Reparation, слайд №8Tissue repair. Regeneration and Reparation, слайд №9Tissue repair. Regeneration and Reparation, слайд №10Tissue repair. Regeneration and Reparation, слайд №11Tissue repair. Regeneration and Reparation, слайд №12Tissue repair. Regeneration and Reparation, слайд №13
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Слайды и текст этой презентации

Слайд 1


Tissue Repair 212- b group O’RMONOV NE’MATJON
Описание слайда:
Tissue Repair 212- b group O’RMONOV NE’MATJON

Слайд 2


Tissue Repair may start early after tissue damage regeneration by parenchymal cells of the same type reparation replacement by connective tissue (fibrosis) result - scar
Описание слайда:
Tissue Repair may start early after tissue damage regeneration by parenchymal cells of the same type reparation replacement by connective tissue (fibrosis) result - scar

Слайд 3


Regeneration and Reparation regeneration restoration of normal structure and function persistence of supportive „tissue skeleton“ necessary BM of epithelia reticulin frame in liver reparation restoration of normal shape x function is impaired or lost parenchyma replaced by fibrous tissue
Описание слайда:
Regeneration and Reparation regeneration restoration of normal structure and function persistence of supportive „tissue skeleton“ necessary BM of epithelia reticulin frame in liver reparation restoration of normal shape x function is impaired or lost parenchyma replaced by fibrous tissue

Слайд 4


Tissue types permanent nonproliferative in postnatal life neurons (?), cardiomyocytes (?) stable regeneration as response to injury parenchyma – liver, pancreas, renal tubules mesenchymal cells, endothelium labile continuous regeneration from stem cells (self-renewal) hematopoietic cells in bone marrow surface epithelia – skin, oral cavity, vagina, cervix duct epithelia – salivary glands, pancreas, biliary tract mucosas – GIT, uterus, fallopian tubes, urinary bladder
Описание слайда:
Tissue types permanent nonproliferative in postnatal life neurons (?), cardiomyocytes (?) stable regeneration as response to injury parenchyma – liver, pancreas, renal tubules mesenchymal cells, endothelium labile continuous regeneration from stem cells (self-renewal) hematopoietic cells in bone marrow surface epithelia – skin, oral cavity, vagina, cervix duct epithelia – salivary glands, pancreas, biliary tract mucosas – GIT, uterus, fallopian tubes, urinary bladder

Слайд 5


Cell-ECM interactions not only cells! EMC plays important role in healing interstitial matrix – by fibroblasts basement membrane – by fibroblast and epithelium components collagen (18 types) – I, III, IV, V; tensile strength elastin (+ fibrillin) – return to normal structure after stress glycoproteins - adhesion, binding ECM to cells (fibronectin, laminin) proteoglycans and hyalouronans – lubrication (gels)
Описание слайда:
Cell-ECM interactions not only cells! EMC plays important role in healing interstitial matrix – by fibroblasts basement membrane – by fibroblast and epithelium components collagen (18 types) – I, III, IV, V; tensile strength elastin (+ fibrillin) – return to normal structure after stress glycoproteins - adhesion, binding ECM to cells (fibronectin, laminin) proteoglycans and hyalouronans – lubrication (gels)

Слайд 6


Cell-ECM interactions ECM function mechanical support determination of cell polarity control of cell growth maintenance of cell proliferation establishment of tissue microenvironments storage of regulatory molecules
Описание слайда:
Cell-ECM interactions ECM function mechanical support determination of cell polarity control of cell growth maintenance of cell proliferation establishment of tissue microenvironments storage of regulatory molecules

Слайд 7


Replacement of necrotic tissue resorption by macrophages dissolution by enzymes replacement by granulation tissue uniform mechanism irrespective of inicial trigger the same microscopic appearance angiogenesis migration and proliferation of fibroblasts deposition of ECM maturation and reorganization
Описание слайда:
Replacement of necrotic tissue resorption by macrophages dissolution by enzymes replacement by granulation tissue uniform mechanism irrespective of inicial trigger the same microscopic appearance angiogenesis migration and proliferation of fibroblasts deposition of ECM maturation and reorganization

Слайд 8


Granulation tissue new-formed connective tissue, apparent from 3rd day thin-walled capillary vessels fibroblasts loose extracellular matrix stimulation PDGF, VEGF, FGF, TGF, TNF, EGF inhibition INFalfa, prostaglandins, angiostatins control cyclins, cyclin dependent kinases
Описание слайда:
Granulation tissue new-formed connective tissue, apparent from 3rd day thin-walled capillary vessels fibroblasts loose extracellular matrix stimulation PDGF, VEGF, FGF, TGF, TNF, EGF inhibition INFalfa, prostaglandins, angiostatins control cyclins, cyclin dependent kinases

Слайд 9


Granulation tissue pink soft granular appearance richly vascularized highly cellular myxoid matrix inflammatory cells e.g. surface of wounds, bottom of ulcers
Описание слайда:
Granulation tissue pink soft granular appearance richly vascularized highly cellular myxoid matrix inflammatory cells e.g. surface of wounds, bottom of ulcers

Слайд 10


Angiogenesis neovascularization x vasculogenesis (embryonic process only) highly complex phenomenon angiogenic factors (FGF, VEGF) antiangiogenic factors healing, collateral circulation, tumors
Описание слайда:
Angiogenesis neovascularization x vasculogenesis (embryonic process only) highly complex phenomenon angiogenic factors (FGF, VEGF) antiangiogenic factors healing, collateral circulation, tumors

Слайд 11


Fibrosis and Remodeling scar formation fibroblasts myofibroblasts spindle cells of both fibroblastic and smooth muscle phenotype production of collagen fibres wound contraction
Описание слайда:
Fibrosis and Remodeling scar formation fibroblasts myofibroblasts spindle cells of both fibroblastic and smooth muscle phenotype production of collagen fibres wound contraction

Слайд 12


Fibrosis and Remodeling abundant collagen fibres bridging the defect devoid of inflammatory cells reepithelization of surface defect from skin appendages and/or from periphery secondary changes calcification (dystrophic) ossification (metaplastic) remodeling synthesis and degradation of ECM metalloproteinases (MPs), tissue inhibitors of MPs
Описание слайда:
Fibrosis and Remodeling abundant collagen fibres bridging the defect devoid of inflammatory cells reepithelization of surface defect from skin appendages and/or from periphery secondary changes calcification (dystrophic) ossification (metaplastic) remodeling synthesis and degradation of ECM metalloproteinases (MPs), tissue inhibitors of MPs

Слайд 13


Pathological aspects of healing proud flesh (caro luxurians) excessive amount of GT keloid excessive amount of collagen hyaline plaques serous membranes (spleen, pleura)
Описание слайда:
Pathological aspects of healing proud flesh (caro luxurians) excessive amount of GT keloid excessive amount of collagen hyaline plaques serous membranes (spleen, pleura)

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